MYELOID NEOPLASIA Clinical relevance of Wilms tumor 1 gene mutations in childhood acute myeloid leukemia

نویسندگان

  • Iris H. I. M. Hollink
  • Marry M. van den Heuvel-Eibrink
  • Martin Zimmermann
  • Brian V. Balgobind
  • Marielle Alders
  • Andre Willasch
  • Gertjan J. L. Kaspers
  • Jan Trka
  • Andre Baruchel
  • Siebold S. N. de Graaf
  • Ursula Creutzig
  • Rob Pieters
  • Dirk Reinhardt
  • Michel Zwaan
چکیده

1Department of Pediatric Oncology/Hematology, Erasmus MC–Sophia Children’s Hospital, Rotterdam, The Netherlands; 2AML-BFM Study Group, Department of Pediatric Oncology/Hematology, Medical High School, Hannover, Germany; 3Clincial Genetics, Academic Medical Center, Amsterdam, The Netherlands; 4Department of Hematology and Oncology, University Children’s Hospital of Frankfurt, Frankfurt, Germany; 5Department of Pediatric Oncology/Hematology, VU University Medical Center, Amsterdam, The Netherlands; 6Pediatric Haematology/Oncology, Second Medical School, Charles University Prague, Prague, Czech Republic; 7Hematology, Saint-Louis Hospital, Paris, France; 8Department of Pediatric Oncology/Hematology, Radboud University Medical Center, Nijmegen, The Netherlands; and 9AML-BFM Study Group, University Children’s Hospital, Münster, Germany

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Wilms' tumor (WT1) gene mutations occur mainly in acute myeloid leukemia and may confer drug resistance.

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Wilms’ Tumor (WT1) Gene Mutations Occur Mainly in Acute Myeloid Leukemia and May Confer Drug Resistance

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تاریخ انتشار 2009